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  2. I have a clear skin but got skin patches and even herpes simplex on the lip..... something I only had only once before in my life. I agree with the article. I do not know if it was the medication I took or not..... Those chicken pox rashes mentioned in your article above could also mimic shingles. It seems the corona virus may mimic the herpes infections you have had before in your life. I think the HIV piece of information (a herpes piece of info) on the SARS 2 (corona19) may be responsible for it (my hypothesis). Watch out! This virus latches onto the T-cells and breaks down your immune system. It could hide there (similar to any herpes virus) and wait until your immunity is weak and come out again. I got the same illness twice.... so be warned.......
  3. Dermatologists around the world are gathering data on what may be largely overlooked symptoms of COVID-19: skin conditions ranging from rashes to “pseudo-frostbite.” Many viral illnesses—including chickenpox, measles and mononucleosis—are accompanied by telltale skin rashes, often a result of the body’s heightened inflammatory response while fighting off infection. Though more research is needed, a growing number of case reports and preliminary studies suggest SARS-CoV-2, the virus that causes COVID-19, can also affect the skin. In late March, an Italian physician submitted a letter to the editor of the Journal of the European Academy of Dermatology and Venereology, describing skin conditions that affected about 20% of 88 COVID-19 patients analyzed in the Lombardy region of Italy. Most of them developed a red rash on their torsos, while a few suffered hives or blisters resembling chickenpox. Then, in early April, a dermatology organization representing more than 400 French dermatologists issued a statement noting that among probable COVID-19 patients they had seen skin symptoms including hives, red rashes and frostbite-like lesions on the extremities. And finally, in mid-April, in a letter to the editor of the Journal of the American Academy of Dermatology, a group of Italian physicians described a chickenpox-like rash as “a rare but specific COVID-19-associated skin manifestation.”
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  4. Vitamin D is so important for immunity health, as a resistance to cancer, and many other diseases and immunity disorders. It is a sure sign that Jehovah created us to be outside working in a garden and getting dirt on our hands. Unfortunately, after the flood we now get too much damaging UV rays when we remain in the sun too long. After the flood the ages of humans also drastically became less as the UV built up in the atmosphere. Nevertheless, I am sure that 15 minutes of sun per day can keep one healthy and provide the needed vitamin D. Skin cancer in Scandinavian countries are just as high as countries with dangerous sunlight. The reason: Scandinavians are not getting enough vitamin D.
  5. Patterns of Covid-19 Mortality and Vitamin D An Indonesian Study "When controlling for age, sex, and comorbidity, Vitamin D status is strongly associated with COVID-19 mortality outcome of cases."
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    "...the team used human blood stem cells and triggered mutations in the PARN gene that give rise to dyskeratosis congenita. These were then implanted into mice that were treated with the compounds, with the team finding the treatment boosted TERC, restored telomere length in the stem cells and had no ill effects on the rodents. “This provided the hope that this could become a clinical treatment,” says Nagpal. The team will now continue its work in an effort to prove these small molecules are a safe and effective way to apply the brakes to dyskeratosis congenita, other diseases, and possibly aging more broadly. “We envision these to be a new class of oral medicines that target stem cells throughout the body,” Agarwal says. “We expect restoring telomeres in stem cells will increase tissue regenerative capacity in the blood, lungs, and other organs affected in DC and other diseases.”
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    Over the past century, the opinion that Vit-C can be used to treat cancer and viral infection has shown promises and controversies. There are cases where high dose Vit-C has shown benefits. In some cases, there have been no benefits. However, new knowledge regarding the pharmacokinetic properties of Vit-C, and recent preclinical studies, have revived interest in the utilization of high-dose Vit-C for cancer treatment [[132], [133], [134], [135], [136], [137], [138], [139], [140], [141], [142], [143], [144], [145]]. Similar is the case of using IV Vit-C as antiviral, especially for the recent Covid19 [[146], [147], [148], [149], [150]]. It is believed that IV Vit-C has been particularly effective by inhibiting the production of cytokines storm due to Corvid19. Covid19 pneumonia is an extremely rapidly developing disease with a high mortality rate. The main pathogenesis is the acute lung injury that causes ARDS and death. Antioxidants should have a role in the management of these conditions. Appropriate clinical studies and reports demonstrate that a timely administration of high dose IV Vit-C improves the outcome of Covid19 infection. Additional studies detailing the use of IV Vit-C for the treatment of severe Covid19 infected pneumonia are definitively warranted. Covid19 may continue to happen in the future. Since the development of clinically active vaccines or antiviral drugs targeting specific diseases may take a long time to develop, the use of IV Vit-C as a universal agent for ARDS may have benefits behind Covid19. Additional clinical studies of the IV Vit-C and oral VC (such as liposomal-encapsulated VC) targeting other situations through different mechanisms are required to develop as soon as possible.
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    “This has been on people’s radar for quite a while,” he said. “Somebody on a different floor sneezes …The particle can stay airborne long enough to go all the way through the system and then pop out in somebody else’s office.”
  6. 15mg-30mg of Zinc a day, Vitamin B6 as well. However, these things, as is with Vitamins are to boost your immunity system, make it stronger.
  7. I may add that vitamin D3 is the best form to use and one should always use it with vitamin K2. Vitamin K2 counteracts any hardening of the arteries which can come with use of vit D. I write this from memory...... so research he vit K2 connection to D3.
  8. My opinion is that you keep the correct levels of vitamin D in your body. Now a days, due to staying indoors, it is possible that you will develop the deficiency of vitamin D. Get this checked by your doctor and use the correct dose to stay healthy. Here are my notes: Daily 10 microgram or 400 IU/Day Lippincott’s Biochemistry Review 4th Edition. Page 388 Daily 5 mg of cholecalciferol or 200 IU of Vitamin D. However, 800 IU/Day is shown to reduce the incidence of osteoporotic fractures. 800 IU/Day recommended by the Institute of Medicine to maintain bone health. Frank deficiency is when the levels of 25-hydroxycholecalciferol (25OHD) is less than 20 ng/mL (50 mmol/L)
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    ... No consensus that the oral vitamin D supplements are helpful Ergocalciferol from plants is called Vitamin D2 Cholecalciferol from the animal tissue is called Vitamin D3 7 deidro-cholesterol to (UV light) to cholecalciferol (D3) to 25 hydroxylase in liver to 25 OH D to -hydroxycholecalciferol 1-hydroxylase in kidney to 1,25 DiOH D3 Vitamin D Receptors are abbreviated to VDR in the notes. These are cytosolic receptors.
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    ... Deficiency of vitamin D makes people more susceptible to respiratory tract infections. Vitamin D supplement can reduce these infections by 12% Keep in mind, this is for those who are deficient in this vitamin. If you are not deficient, then it will not make a difference. Because, you are already good. Evidence is not sufficient to say that vitamin D helps prevent respiratory infections. 2018 Study
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    ... People with autoimmune diseases commonly have hypovitaminosis D What are cathelicidins?
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    ... What are beta defensins?
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    _... 2011 Study
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    ... Activation of TLR and PAMPs on macrophages increases the production of 1, alpha hydroxylase and the VDR. This in turn causes more production of cathelicidine and beta defensins 4. Vitamin D also affects monocytes to reduce production of IL1, IL6, IL8, IL12, and TNF. Regulating the immune response. Vitamin D acts on the dendritic cells to inhibit/reduce their differentiation and maturation. It reduces their MHCII expression, co-stimulatory molecules like CD40, CD80, CD86, decreased production of IL-12 etc. 2013 Study
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    ... Vitamin D improves immune system by helping produce cathelicidine and by helping modulate inflammatory cascade. Especially for the respiratory infections. It also helps produce NFk-B (nuclear factor kappa light chain for activated B cells.) ( Notes about the NFkB
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    Helps with cytokine production and cell survival. NFk-B factor becomes active when T and B cell receptors become active. Its activation causes the gene expressions that allow T cell development, maturation, and proliferation. T and B cells have Vitamin D receptors that help them produce NFk-B ) Continuing the notes from the study: In pulmonary infections. Lung epithelial cells are able to convert inactive vitamin D to its active form. This active Vitamin D then helps produce cathelicidine. Which helps kill the pathogens. Vitamin D seems to favor TH2 and TH17 cell activation which is anti-inflammatory profile. Vitamin D seems to reduce the activation of TH1 cells which are pro inflammatory by their IFN-gamma action. A deficiency of vitamin D may dysregulate the inflammatory response of the body. Vitamin B also increases the IkBa. This leads to inhibition of NFkB. This helps reduce the production of IL6 and IL8 by inhibiting the production of NFkB.
  9. New Coronavirus (COVID-19) Drugs: Remdesivir, Tocilizumab (Actemra), and Leronlimab | New Coronavirus Treatment Remdesivir, Tocilizumab, and Leronlimab are new medications against Coronavirus (COVID-19) that are being explored. Besides hydroxychloroquine, these are other medications being investigated in the context of a clinical trial and some for compassionate use to treat coronavirus (COVID-19). Remdesivir, developed by Gilead Sciences Inc., is an investigational broad-spectrum antiviral treatment that was previously tested in humans with Ebola and had shown promise in animal models for treating Middle East respiratory syndrome (MERS) and severe acute respiratory syndrome (SARS), which are caused by other coronaviruses. Currently Gilead Sciences has made Remdesivir available for coronavirus clinical trials and to more than 1800 people on a compassionate use basis. The hope is that the results from the phase 3 clinical trials will be available by the end of May, 2020. Tocilizumab is a monoclonal antibody that competitively inhibits the binding of interleukin-6 (IL-6) to its receptor (IL-6R). And by inhibiting the entire receptor complex, you block the signal to induce more inflammation from coronavirus. In a recent study from China, researchers noted Tocilizumab appears to be an effective treatment option in COVID‐19 patients with a risk of cytokine storms. And for these critically ill patients with elevated IL‐6, the repeated dose of the Tocilizumab is recommended. Leronlimab, made by the Canadian pharmaceutical Cytodyn, is a CCR5 reecptor inhibitor drug that was developed to treat HIV. The CCR5 receptor stands for “chemokine receptor type 5” and is expressed on t-cells, macrophages and even on breast and prostate cancer cells. By blocking this receptor, the principle is that you are block cytokines that direct cell movement for coronavirus (COVID-19). And by blocking the movement, you don’t bring immune cells to the site of infection from coronavirus (COVID-19), thus decreasing the risk of a cytokine storm. While there are many more drugs still in the pipeline to treatment COVID-19, these are the new drugs I feel have the most promise and are moving along to phase 3 clinical trials and onto the consumer market. - Dr. Yo.
  10. So cool.... inflammatory bowel diseases could be treated by introducing these “missing” acids into the colon. Dr Aida Habtezion, a co-author of the study from Stanford University, told the Guardian the findings suggest a new approach to treating ulcerative colitis could be developed, based on introducing microbes or the substances they produce. Many patients find current treatments do not work or have side-effects. “I think it brings another mechanism, a natural way, in which we can reconstitute and hopefully treat our patients,” Habtezion said. Writing in the journal Cell Host & Microbe, the team report how they studied two groups of patients who had had their colons removed and pouches created. For one group, of 17 patients, this surgery was to treat ulcerative colitis whereas for the other group, of seven patients, it was because of a different condition that does not involve inflammation. Certain bacteria in the gut are known to convert bile acids, produced by the liver, into other substances, called secondary bile acids – these substances have recently been suggested to have an anti-inflammatory role.
  11. Chest CT images of COVID-19 lung involvement in a 44-year old Huanan Seafood worker. Day 13 of symptom progression (died 7 days later) The patches are (predominantly) diffuse alveolar damage. That leads to edema (a fluid build up in the lungs,) and the beginnings of fibrosis (excess connective tissue.) Due to all of the replication and damage going on, there is also a build up up extra proteins and dead cells which clog up the lungs.
  12. Patient on days 8 and 11 after the onset of illness: Full report:
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    Heres a normal one for comparison: Here is one showing pneumonia in the right lung base: So basically it clogs up the lungs with the dense white stuff The dense white stuff being fluid, mucus, and other stuff that generally shouldn’t be in your lungs. This is what all cases of pneumonia do, regardless of their origin (viral, bacterial, chemical, etc.) and relative severity. How might the treatment in a hospital be for this? They install apparatuses to aid breathing and create an environment in the body that counters inflammation. They basically try to support the bodies immune system as best as possible and try help the patient to breathe as best as possible. In case of the Virus you need to take additional steps to make it harder for the pathogens to survive which in case of the Corona would be an alkaloid gastrointestinal environment as well as an increased PH value (which is hard to accomplish in a short time period).
  13. The United States has confirmed the first case of the new coronavirus on its territory. The Centers for Disease Control said the virus, which originated in China, had been diagnosed in a US resident who arrived in Seattle from China. The virus, which spread from the Chinese city of Wuhan, has infected almost 300 people, and six have died. North Korea has temporarily closed its borders to foreign tourists in response to the threat, a tour operator says. The patient diagnosed in the US - reported to be a man in his 30s - returned from Wuhan on 15 January, the CDC said. "The patient sought care at a medical facility in the state of Washington, where the patient was treated for the illness," it added in a statement. "Based on the patient's travel history and symptoms, healthcare professionals suspected this new coronavirus." Laboratory testing of a clinical specimen confirmed the diagnosis on 20 January, the CDC statement continued.
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  14. The irony of it all, there were people who spoken loudly regarding this issue, and it is only years later, now people are concerned for the risks of vaping. The parents and the community needs to teach and educate their kids, and older ones who have kids concerning the issue to reduce more young ones entering into the realm of vaping. Solutions to the problem is always key to a sure victory.
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    • Isabella

      Good ideas 
       

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    • 4Jah2me  »  Srecko Sostar

      Hi Srecko. I hope you can see this photo. This is my daily driving car. It is outside a Dance Studio where  I have danced and hope to go dancing again, John 

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    • Tennyson  »  Queen Esther

      Hello my sister, i have not head from you long sice. I hope you are wel. Hope to hear from you soon. Agape.
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    • Doryseeker  »  4Jah2me

      *** it-2 p. 7 Jehovah ***
      The Codex Leningrad B 19A, of the 11th century C.E., vowel points the Tetragrammaton to read Yehwahʹ, Yehwihʹ, and Yeho·wahʹ. Ginsburg’s edition of the Masoretic text vowel points the divine name to read Yeho·wahʹ. (Ge 3:14, ftn) Hebrew scholars generally favor “Yahweh” as the most likely pronunciation. They point out that the abbreviated form of the name is Yah (Jah in the Latinized form), as at Psalm 89:8 and in the expression Ha·lelu-Yahʹ (meaning “Praise Jah, you people!”). (Ps 104:35; 150:1, 6) Also, the forms Yehohʹ, Yoh, Yah, and Yaʹhu, found in the Hebrew spelling of the names Jehoshaphat, Joshaphat, Shephatiah, and others, can all be derived from Yahweh. Greek transliterations of the name by early Christian writers point in a somewhat similar direction with spellings such as I·a·beʹ and I·a·ou·eʹ, which, as pronounced in Greek, resemble Yahweh. Still, there is by no means unanimity among scholars on the subject, some favoring yet other pronunciations, such as “Yahuwa,” “Yahuah,” or “Yehuah.”
      Since certainty of pronunciation is not now attainable, there seems to be no reason for abandoning in English the well-known form “Jehovah” in favor of some other suggested pronunciation. If such a change were made, then, to be consistent, changes should be made in the spelling and pronunciation of a host of other names found in the Scriptures: Jeremiah would be changed to Yir·meyahʹ, Isaiah would become Yeshaʽ·yaʹhu, and Jesus would be either Yehoh·shuʹaʽ (as in Hebrew) or I·e·sousʹ (as in Greek). The purpose of words is to transmit thoughts; in English the name Jehovah identifies the true God, transmitting this thought more satisfactorily today than any of the suggested substitutes.
      *** it-2 p. 7 Jehovah ***
      The Codex Leningrad B 19A, of the 11th century C.E., vowel points the Tetragrammaton to read Yehwahʹ, Yehwihʹ, and Yeho·wahʹ. Ginsburg’s edition of the Masoretic text vowel points the divine name to read Yeho·wahʹ. (Ge 3:14, ftn) Hebrew scholars generally favor “Yahweh” as the most likely pronunciation. They point out that the abbreviated form of the name is Yah (Jah in the Latinized form), as at Psalm 89:8 and in the expression Ha·lelu-Yahʹ (meaning “Praise Jah, you people!”). (Ps 104:35; 150:1, 6) Also, the forms Yehohʹ, Yoh, Yah, and Yaʹhu, found in the Hebrew spelling of the names Jehoshaphat, Joshaphat, Shephatiah, and others, can all be derived from Yahweh. Greek transliterations of the name by early Christian writers point in a somewhat similar direction with spellings such as I·a·beʹ and I·a·ou·eʹ, which, as pronounced in Greek, resemble Yahweh. Still, there is by no means unanimity among scholars on the subject, some favoring yet other pronunciations, such as “Yahuwa,” “Yahuah,” or “Yehuah.”
      Since certainty of pronunciation is not now attainable, there seems to be no reason for abandoning in English the well-known form “Jehovah” in favor of some other suggested pronunciation. If such a change were made, then, to be consistent, changes should be made in the spelling and pronunciation of a host of other names found in the Scriptures: Jeremiah would be changed to Yir·meyahʹ, Isaiah would become Yeshaʽ·yaʹhu, and Jesus would be either Yehoh·shuʹaʽ (as in Hebrew) or I·e·sousʹ (as in Greek). The purpose of words is to transmit thoughts; in English the name Jehovah identifies the true God, transmitting this thought more satisfactorily today than any of the suggested substitutes.
       
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